Acta Cancerológica                      Vol. XXXI   Nº 1   Mayo 2002

 

REFERENCIAS BIBLIOGRÁFICAS

1. Anguita E., Villegas A., Diaz-Mendiavilla J., Gonzales A., Del Potro E., Espinós D. 1998. Detection of bcr/abl mRNA in a case of chronic myelogenous leukemia in long-term remission: CML or sensitivity of detection?. Haemat. 83: 744-747.

2. Au WY, Lie AK, Ma SK, Wan TS, Liang R, Leung YH, Kwong YL. 2001. Philadelphia (Ph) chromosome-positive chronic myeloid leukemia relapsing as Ph-negative leukemia after allogenic bone marrow transplantation. Br L Haematol 114(2):365-368.

3. Bartram C., Klein A., Hagemeijer A. 1983. Translocation of the c-abl oncogene correlate whit the presence of the Ph chromosome CML. Nature (Lond) 306: 277-280.

4. Crose C.M., Huebner K., Isobe M. 1987. Mapping of four distinct BCR related loci to chromosome: order of BCR loci relative to CML and ALL breakpoints. Proc. Natl. Acad Sc¡. USA. 84: 7174-7178.

5. Fioretos T. 1996. Molecular genetic studies of Ph-positive leukemias and the Bcr and Abl genes. Clin Genet 2: 21.

6. Groffen J., Stephenson J., Heisterkamp N. 1984. Philadelphia chromosomal breakpoints are clustered within a limited region, bcr, one chromosome 22. Cell 36: 93-99.

7. Groffen J., Heisterkamp N. 1987. The BCR/ABL hybrid gene. Baillieres Clin Haematol 1: 983-999.

8. Hagemeijer A. 1987. Chromosome abnormalities in CML. Baillieres Clin Haematol 1: 963-981.

9. Kitzis A., Brizard F., Dascalescu C., Chomel JC., Guilhot F., Brizard A. 2001. Persistence of transcriptionally silent BCR-ABL rearrangement in chronic myeloid leukemia patients in sustained complete cytogenetic remission. Leuk Lymphoma 42(5):933-944.

10. Kurzrock R., Gutterman J., Talpaz M. 1988. The molecular genetics of Ph+ leukemia. N. Engl. J. Med. 319: 990-998.

11. Radich JP., Gooley T., Bryant E., Chauncey T., Clift R., Beppu L., Edmans S., Flowers ME., Kerkof K., Nelson R., Appelbaum FR. 2001. The significance of bcr-abl molecular detection in chronic myeloid leukemia patients "late" 18 months or more after transplantation. Blood 98(6): 1701-1707.

12. Santucci M., Saglio G., Tura S. 1996. Pathogenesis and progression of chronic myeloid leukemia. Haemat 81: 63-76.

13. Sun B., Jiang G., Zaydan MA., La Russa VF., Safah H., Ehrlich M. 2001. ABL1 promoter methylation can exist independently of BCR-ABL transcription in chronic myeloid leukemia hematopoietic progenitors. Cancer Res 61(18): 6931-6937.

14. Van Etten R., Jackson R., Baltimore D. 1989. The mouse type 4 c-abl product is a nuclear protein, and activation of transforming ability is associated with cytoplasmic localization. Cell 58:669-678.

15. Vickers M. 1996. Estimation of the number of mutation necessary to cause chronic myeloid leukemia from epidemiological data. Br J Haemat 94: 1-4.


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